First anniversary of Molecular Cancer: achievements and future goals
© Deora et al; licensee BioMed Central Ltd. 2003
Received: 11 May 2003
Accepted: 30 July 2003
Published: 30 July 2003
Molecular Cancer has been launched for one year now. Here, we describe achievements of the past year and future goals of the journal.
It is a time of change . There is no doubt that the uncensored exchange of scientific results hastens progress and will be the gold standard for any journal [2–7]. BioMed Central, the publisher of Molecular Cancer, is passionately committed to maximizing the visibility and impact of published papers by open-access publishing. In addition, our online submission and processing system allows that manuscripts are quickly reviewed, revised, re-reviewed and published without moving a piece of paper.
Manuscripts submitted for publication must meet the journal's criteria for acceptance. Each manuscript must have a foundation of well-developed, solid data plus a significant advance over existing literature. Pre-submission inquiries will be screened and authors receive a response no later than one day after receiving the request. After passing initial screening, the manuscript will be subjected to rigorous and anonymous peer-review by three or more scientists. Associate and deputy Editors serve as core referees. The authors are informed of the comments made on their paper without delay. A few examples may illustrate how quickly papers are reviewed and published. The paper by Nawaz et al  may be one example. Hours after receiving the paper, we sent it out to three referees outside the editorial board and revised by the authors. Finally, we accepted the paper on the following day. This achievement is due to on-line processing system that the journal employs. Molecular Cancer published a number of pivotal research papers in its first year. The readers frequently accessed number of the published papers. For example, the paper by Zhang et al  has been accessed over 3000 times. This probably would not have been achieved if access had been restricted by subscription.
Four most accessed papers of all time published by Molecular Cancer. Data as of July 22, 2003
During my time as editor of Molecular Cancer, I received many encouraging e-mails from fellow readers and contributors. Readers appreciate the open-access policy of our publisher. In addition, it is positively noted that large amounts of data appear on the journal site . Last but not least, every paper published by Molecular Cancer is available through PubMed.
Molecular Cancer publishes manuscripts in all areas of cancer-related science. Papers are be subjected to fast, fair, rigorous and anonymous peer-review. Once, a paper passed the peer-review process, it appears immediately as just published manuscript PDF followed by a fully formatted PDF and web version. All published papers are indexed in PubMed and archived in PubMed Central.
CS drafted the paper, ABD, KL and GMS provided comments, critique and suggestions for its improvement. All authors read and approved the final manuscript.
CS acknowledges support from the German Research Foundation. GMS is a recipient of a Fellowship of the Cancer League of Bern, Switzerland.
- Wylie C: A time of change. Development. 2002, 129: 5552-10.1242/dev.00208.View Article
- Atlas R, Campbell P, Cozzarelli NR, Curfman G, Enquist L, Fink G, Flanagin A, Fletcher J, George E, Hammes G, Heyman D, Inglesby T, Kaplan S, Kennedy D, Krug J, Levinson RE, Marcus E, Metzger H, Morse SS, O'Brien A, Onderdonk A, Poste G, Renault B, Rich R, Rosengard A, Salzburg S, Scanlan M, Shenk T, Tabor H, Varmus H, Wimmer E, Yamamoto K: Uncensored exchange of scientific results. Proc Natl Acad Sci USA. 2003, 100: 1464- 10.1073/pnas.0630491100View ArticlePubMed
- Cozzarelli NR: Unfettered access to published results. Proc Natl Acad Sci USA. 2001, 98: 8159- 10.1073/pnas.171309098PubMed CentralView ArticlePubMed
- Cozzarelli NR: For free access, follow the brick red buttons. Science. 2001, 292: 51-52. 10.1126/science.292.5514.51cView ArticlePubMed
- Luttich K, Schmidt C, Sclabas G M.: Frequently asked questions about Molecular Cancer. Mol. Cancer. 2003, 2: 16- 10.1186/1476-4598-2-16PubMed CentralView Article
- Till JE: Success factors for open access. J. Med. Internet Res. 2003, 5: E1-PubMed CentralView ArticlePubMed
- Newmark P: Peer review and the rewards of open access. Nature. 2003, 422: 661- 10.1038/422661bView ArticlePubMed
- Schmidt C: Bench and Bedside. Mol. Cancer. 2002, 1: 1- 10.1186/1476-4598-1-1PubMed CentralView Article
- Gao X, Loggie BW, Nawaz Z: The roles of sex steroid receptor coregulators in cancer. Mol. Cancer. 2002, 1: 7- 10.1186/1476-4598-1-7PubMed CentralView ArticlePubMed
- Zhang P, Gao WY, Turner S, Ducatman BS: Gleevec (STI-571) inhibits lung cancer cell growth (A549) and potentiates the cisplatin effect in vitro. Mol Cancer. 2003, 2: 1- 10.1186/1476-4598-2-1PubMed CentralView ArticlePubMed
- Kunz M, Ibrahim SM: Molecular responses to hypoxia in tumor cells. Mol. Cancer. 2003, 2: 23- 10.1186/1476-4598-2-23PubMed CentralView ArticlePubMed
- Vasseur S, Malicet C, Calvo EL, Labrie C, Berthezene P, Dagorn JC, Iovanna JL: Gene expression profiling by DNA microarray analysis in mouse embryonic fibroblasts transformed by rasV12 mutated protein and the E1A oncogene. Mol Cancer. 2003, 2: 19- 10.1186/1476-4598-2-19PubMed CentralView ArticlePubMed
- Lynch C, Tycko B, Bestor T, Walsh C: Reactivation of a silenced H19 gene in human rhabdomyosarcoma by demethylation of DNA but not by histone hyperacetylation. Mol Cancer. 2002, 1: 2- 10.1186/1476-4598-1-2PubMed CentralView ArticlePubMed
- Delsite R, Kachhap S, Anbazhagan R, Gabrielson E, Singh KK: Nuclear genes involved in mitochondria-to-nucleus communication in breast cancer cells. Mol Cancer. 2002, 1: 6- 10.1186/1476-4598-1-6PubMed CentralView ArticlePubMed
- Carew JS, Huang P: Mitochondrial defects in cancer. Mol Cancer. 2002, 1: 9- 10.1186/1476-4598-1-9PubMed CentralView ArticlePubMed
- Narayan G, Arias-Pulido H, Koul S, Vargas H, Zhang FF, Villella J, Schneider A, Terry MB, Mansukhani M, Murty VV: Frequent Promoter Methylation of CDH1, DAPK, RARB, and HIC1 Genes in Carcinoma of Cervix Uteri: Its Relationship to Clinical Outcome. Mol Cancer. 2003, 2: 24- 10.1186/1476-4598-2-24PubMed CentralView ArticlePubMed
- Wary K, Thakker G, Humtsoe J, Yang J: Analysis of VEGF-responsive Genes Involved in the activation of endothelial cells. Mol Cancer. 2003, 2: 25- 10.1186/1476-4598-2-25PubMed CentralView ArticlePubMed
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