Fig. 3From: MicroRNA-505 functions as a tumor suppressor in endometrial cancer by targeting TGF-αmiR-505 overexpression induces G1 phase arrest and promotes apoptosis of endometrial carcinoma cells. MiR-505 transfection significantly induced G1 arrest (a) and elevated apoptosis (b) compared with the control and mock cells. Results are representative of three separate experiments; data are expressed as the mean ± standard deviation, *P < 0.05Back to article page