Skip to main content
Fig. 1 | Molecular Cancer

Fig. 1

From: Bit1 knockdown contributes to growth suppression as well as the decreases of migration and invasion abilities in esophageal squamous cell carcinoma via suppressing FAK-paxillin pathway

Fig. 1

Bit1 protein expression in many different ESCC cell lines and normal esophageal epithelial cell Het-1A. Total protein was extracted from various ESCC cell lines such as EC9706, Eca109, TE1, TE13, KYSE-450 and KYSE-70 as well as Het-1A, and Western blotting was utilized to investigate the Bit1 protein level in ESCC cell lines above. a Western blotting assay for Bit1 protein expression in ESCC cell lines including EC9706, Eca109, TE1, TE13, KYSE-450 and KYSE-70 as well as Het-1A, and β-actin was used as internal control. b The relative Bit1 protein level was counted by the ratio of Bit1 level to β-actin level, and the results from the three independently repeated experiments, expressed as means ± SD, were investigated by SPSS 17.0 statistical software, *P < 0.05, compared with EC9706, Eca109, TE13, and KYSE-70, #P < 0.01, compared with the other ESCC cell lines, **P < 0.01, compared with all of ESCC cell lines

Back to article page

Molecular Cancer

ISSN: 1476-4598