Fig. 6From: Overexpression of G protein-coupled receptor GPR87 promotes pancreatic cancer aggressiveness and activates NF-κB signaling pathwayClinical relevance of GPR87-induced NF-κB activation in human pancreatic cancer. Expression analysis (left) and correlation (right) of GPR87 expression and BCL-xL, CCND1, VEGF-C and nuclear p65 expression in 10 freshly collected human pancreatic tumor samples (T); α-Tubulin and the nuclear protein p84 were used as loading controls. Each bar represents the mean ± SD of three independent experimentsBack to article page