Fig. 2From: Characterization of MED12, HMGA2, and FH alterations reveals molecular variability in uterine smooth muscle tumorsMutual exclusivity of MED12 mutations, HMGA2 overexpression, and biallelic FH inactivation. Representative figures of uterine smooth muscle tumors with different molecular genetic features. a MED12 mutation-positive conventional leiomyoma, b cellular leiomyoma with HMGA2 overexpression, c 2SC-positive leiomyoma with bizarre nuclei, and d leiomyosarcoma without the known genetic leiomyoma driver aberrations. Antibody stainings are shown with ×40 magnificationBack to article page