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Table 1 EVs-related specific chemokines in tumor environment

From: Microvesicles and chemokines in tumor microenvironment: mediators of intercellular communications in tumor progression

Tumor type

EVs type

Content of chemokines

Role of Evs

References

Breast cancer

MVs

CCL5, CCR6

metastasis, proliferation and cancer cell-induced angiogenesis

[5, 47]

Colorectal adenocarcinoma

MVs

CCR6, CX3CR1, CCL2

antiapoptotic effect on monocytes, AKT kinase activation,proliferation

[38, 49]

Gastric cancer

MVs

CCR6

tumor progression

[52]

Glioblastoma

EVs

CCR8, CCL18

proliferation and drug resistance

[56]

Haemophilia A

MVs

CXCL8, CXCL10, CCL2

immunological profile

[36]

Leukemia

Apoptotic blebs

CCR7, CCL19

immuno modulation

[50]

 

Exsomoes

CCL3, CCL4, CXCR4

cell survival and drug resistance

[74]

 

MVs

CXCR4, CCL12

cell survival

[68]

Lung carcinoma

MVs

CCR6, CX3CR1, CXCR4

metastasis, antiapoptotic effect on monocytes, AKT kinase activation

[38, 58]

 

Exsomoes

CCL2, CCL3, CCL4, CCL5, CCL20

antitumor immune response

[51]

Lymphoma

Exosomes

CCL2, CCL7

macrophage recruitment

[42]

Melanoma

Exosomes

CCL2, CCL3, CCL4, CCL5, CCL7, CCL20

macrophage recruitment, antitumor immune response

[42, 51]

 

MVs

CCL2

inhibi ttumor proliferation

[49]

Pancreatic adenocarcinoma

MVs

CCR6, CX3CR1, CCL2, CCL3, CCL4, CCL5, CXCL8

anti-apoptosis, AKT kinase activation, angiogenesis

[38, 39]

Prostate cancer

Oncosomes

CXCL12

oncogenic signaling

[23]