Fig. 3From: PIK3CA hotspot mutations differentially impact responses to MET targeting in MET-driven and non-driven preclinical cancer modelsPresence of PIK3CA mutations confers resistance in colony-forming assays and wound-healing upon MET inhibition. a, colony-forming assays (left: relative colony quantification; right: representative images). b, wound-healing assays. Cell contours were enhanced for clarity using the Paint.NET software. P-values represent comparisons between treatment conditions and vehicle-treated controlsBack to article page