Fig. 9From: METTL14-mediated m6A modification of circORC5 suppresses gastric cancer progression by regulating miR-30c-2-3p/AKT1S1 axisSchematic representation of proposed mechanism of METTL14 in GC. METTL14 increased the m6A levels of circORC5 and inhibited circORC5 expression, thereby upregulated miR-30c-2-3p and downregulated AKT1S1 and EIF4AB, contributing to suppression of GC progressionBack to article page