Open Access

Erratum to: Alpha-enolase as a potential cancer prognostic marker promotes cell growth, migration, and invasion in glioma

Contributed equally
Molecular Cancer201513:235

https://doi.org/10.1186/1476-4598-13-235

Received: 16 October 2014

Accepted: 16 October 2014

Published: 20 January 2015

The original article was published in Molecular Cancer 2014 13:65

Correction

After the publication of this work [1] it was brought to the authors’ attention that the U251-pLVTHM panel in Figure fiveB and the U251 negative control (NC) panel in Figure fiveD contained a duplication in error. The correct version of Figure five (Figure 1 here) is given below.
Figure 1

Stably inhibited ENO1 expression decreases cell migration and invasion. (A). Stablydownregulating ENO reduced the migration ability of shENO1-U251 and shENO1-U87 cells in vitro. (B). Stably suppressed ENO1 reduced in vitro invasion of shENO1-U251 and shENO1-U87 cells. (C). Transiently downregulated ENO1 dramatically decreased the migration ability of U251 and U87 cells in vitro. (D). Transiently suppressed ENO1 inhibited in vitro invasion of U251 and U87 cells. Data were presented were presented as mean ± SD for three independent experiments. *P < 0.05, statistically significant difference.

The authors regret any inconvenience that this inaccuracy may have caused.

Notes

Declarations

Authors’ Affiliations

(1)
Department of Neurosurgery, Nanfang Hospital, Southern Medical University
(2)
Cancer Research Institute of Southern Medical University
(3)
Department of Neurosurgery, Affiliated Hospital, Youjiang Medical College for Nationalities
(4)
Department of Pathology, Basic School of Guangzhou Medical College

References

  1. Song Y, Luo Q, Long H, Hu Z, Que T, Zhang X, Li Z, Wang G, Yi L, Liu Z, Fang W, Qi S: Alpha-enolase as a potential cancer prognostic marker promotes cell growth, migration, and invasion in glioma. Mol Cancer. 2014, 13: 65- 10.1186/1476-4598-13-65PubMed CentralView ArticlePubMedGoogle Scholar

Copyright

© Song et al.; licensee BioMed Central. 2014

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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