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Erratum to: Involvement of GTA protein NC2beta in Neuroblastoma pathogenesis suggests that it physiologically participates in the regulation of cell proliferation
Molecular Cancer volume 7, Article number: 59 (2008)
The Original Article was published on 06 June 2008
After publication of this work [1], we noticed that as Dr Rosario Angelica was added as an author late in the process, details were inadvertently omitted from the "Authors' contributions" and "Competing interests" sections of the article. These have now been modified accordingly.
References
Di Pietro Cinzia, Ragusa Marco, Barbagallo Davide, Duro Laura, Guglielmino Maria, Majorana Alessandra, Giunta Veronica, Rapisarda Antonella, Tricarichi Elisa, Miceli Marco, Grillo Agata, Banelli Barbara, Defferari Isabella, Forte Stefano, Laganà Alessandro, Bosco Camillo, Giugno Rosalba, Pulvirenti Alfredo, Ferro Alfredo, Grzeschik Karl, Di Cataldo Andrea, Tonini Gian, Romani Massimo, Purrello Michele: Involvement of GTA protein NC2beta in Neuroblastoma pathogenesis suggests that it physiologically participates in the regulation of cell proliferation. Molecular Cancer. 2008, 7: 52- 10.1186/1476-4598-7-52
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The authors declare that they have no competing interests.
Authors' contributions
MP conceived and directed the project. CdP, MR (Ge), GPT, AdC, KHG, AF, AG designed some of the experiments. MR (CT), DB, LRD, MRG, AM, RA, VG, AR, ET, MM, BB, ID, SF, AL, CB, RG, AP carried out experiments. MP and MR (Ge) wrote the paper.
The online version of the original article can be found at 10.1186/1476-4598-7-52
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Open Access This article is published under license to BioMed Central Ltd. This is an Open Access article is distributed under the terms of the Creative Commons Attribution License ( https://creativecommons.org/licenses/by/2.0 ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
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Di Pietro, C., Ragusa, M., Barbagallo, D. et al. Erratum to: Involvement of GTA protein NC2beta in Neuroblastoma pathogenesis suggests that it physiologically participates in the regulation of cell proliferation. Mol Cancer 7, 59 (2008). https://doi.org/10.1186/1476-4598-7-59
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DOI: https://doi.org/10.1186/1476-4598-7-59